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Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

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Record title

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

Record identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4255242

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

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https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4255242

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

Full title

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

Publisher

England: Nature Publishing Group

Journal title

Nature (London), 2014, Vol.515 (7527), p.431-435

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4255242

Language

English

Formats

Publication information

Publisher

England: Nature Publishing Group

More information

SCOPE AND CONTENTS

Contents

Ischaemia-reperfusion injury occurs when the blood supply to an organ is disrupted and then restored, and underlies many disorders, notably heart attack and stroke. While reperfusion of ischaemic tissue is essential for survival, it also initiates oxidative damage, cell death and aberrant immune responses through the generation of mitochondrial rea...

ALTERNATIVE TITLES

Full title

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS

Identifiers

PRIMARY IDENTIFIERS

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4255242

Permalink

https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4255242

OTHER IDENTIFIERS

ISSN

0028-0836

E-ISSN

1476-4687

DOI

10.1038/nature13909

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