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Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation...

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Record title

Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice

Record identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6086584

Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice

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https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6086584

Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice

Full title

Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice

Publisher

Ireland: Elsevier B.V

Journal title

Cancer letters, 2018, Vol.423, p.71-79

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6086584

Language

English

Formats

Publication information

Publisher

Ireland: Elsevier B.V

More information

SCOPE AND CONTENTS

Contents

PanINs and IPMNs are the two most common precursor lesions that can progress to invasive pancreatic ductal adenocarcinoma (PDA). DCLK1 has been identified as a biomarker of progenitor cells in PDA progressed from PanINs. To explore the potential role of DCLK1-expressing cells in the genesis of IPMNs, we compared the incidence of DCLK1-positive cell...

ALTERNATIVE TITLES

Full title

Pancreatic DCLK1+ cells originate distinctly from PDX1+ progenitors and contribute to the initiation of intraductal papillary mucinous neoplasm in mice

Identifiers

PRIMARY IDENTIFIERS

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6086584

Permalink

https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6086584

OTHER IDENTIFIERS

ISSN

0304-3835

E-ISSN

1872-7980

DOI

10.1016/j.canlet.2018.03.009

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