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BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling path...

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Record title

BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling pathway in glomerular mesangial cells

Record identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6947818

BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling pathway in glomerular mesangial cells

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https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6947818

BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling pathway in glomerular mesangial cells

Full title

BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling pathway in glomerular mesangial cells

Publisher

Greece: Spandidos Publications UK Ltd

Journal title

Molecular medicine reports, 2020, Vol.21 (2), p.795-805

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6947818

Language

English

Formats

Publication information

Publisher

Greece: Spandidos Publications UK Ltd

More information

SCOPE AND CONTENTS

Contents

The aim of the present study was to investigate the involvement of B cell‑activating factor (BAFF) in the pathogenesis of IgA nephropathy by activating the tumor necrosis factor receptor‑associated factor 6 (TRAF6)/NF‑κB signaling pathway in glomerular mesangial cells. For the clinical analysis, blood, urine and kidney tissue samples were collected...

ALTERNATIVE TITLES

Full title

BAFF is involved in the pathogenesis of IgA nephropathy by activating the TRAF6/NF‑κB signaling pathway in glomerular mesangial cells

AUTHORS, ARTISTS AND CONTRIBUTORS

Identifiers

PRIMARY IDENTIFIERS

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6947818

Permalink

https://collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6947818

OTHER IDENTIFIERS

ISSN

1791-2997

E-ISSN

1791-3004

DOI

10.3892/mmr.2019.10870

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